Revision TKR-Methods for Establishing Equal Flexion & Extension Gaps

 

Kelly G. Vince, M.D., F.R.C.S.(C)

Assistant Clinical Professor

University of California - Irvine

Irvine, California

Associate Surgeon,

Kerlan-Jobe Orthopedic Clinic,

Los Angeles, California

General Rules for equalizing flexion and extension gaps:

 

*    Throw away the spacer blocks. They are based on having normal amounts of bone.

*    Use trial components.

*    Forget the tibia. What is missing from the tibia has no kinematic implication, so long as you have enough bone for fixation and to eliminate recurvatum.

*    The femur is where it’s happening

*    Key to the flexion gap à POSTERIOR FEMORAL SURFACE

*    Key to the extension gapà DISTAL FEMORAL SURFACE

*    Instability results more frequently from bone loss than ligament loss

*    You really only need an MCL and an extensor mechanism

*    Experiment with ligament advancements

*    Use constrained components infrequently- know when and why you want to use it

*    Always go to surgery with constrained components available

*    Never go to surgery planning to use constrained components

 

Three Step Plan for Revision TKR

Step 1 Reestablish the Tibial Platform

The tibial surface comprises both the flexion and extension gaps and is the natural starting point for reconstruction. When residual tibial bone is adequate, standard components may be implanted. However, when the proximal tibia is osteopenic or has defects that must be rebuilt, it is frequently necessary to augment fixation with intramedullary stems. If this option is selected, the medullary canal must be opened.

The position and orientation of the intra-medullary rod will determine the position of the component attached to it. A tibial rod must enter the proximal tibia at a point over the center of the diaphysis, which may not correspond to the center of the cut surface of the proximal tibia. If the rod is not oriented parallel to the mechanical axis of the tibia, then only a smaller rod will fit, and malalignment will ensue.

The diameter of the intramedullary canal should be determined from preoperative radiographs. With these dimensions in mind, the size of the canal can be probed with reamers-and active reaming of the tibia should be minimized. Assemble and insert a trial tibial component with an intramedullary rod. Ignore tibial defects, but confirm the alignment and  posterior slope. The articular platform should not slope anteriorly.

The trial component will help define the defect in the proximal tibia. If there  is contact between the trial and bone circumferentially, then any defects are "contained" and can be filled with bone cement or particulate bone graft. If the angle of the proximal tibial bone does not match the undersurface of the tibial component, the proximal tibia should be sawn to an angle that corresponds to an available augment if this can be done with minimal loss of bone. Attach the augment to the trial and reinsert.

Do not reconstruct tibial defects yet, but keep the trial tibial component in place to protect the bone. Complete a general plan for balancing gaps and selecting component sizes and positions first, before attending to details such as reconstructing bone defects.


 

Bone Defects

 

DEFECT

SOLUTION

- Small Contained

Cement or particulate graft

- Non contained

Modular Blocks, wedges or custom devices

 

 

 

Step 2 Stabilize the Knee in Flexion

Summary

 

Goals

SOLUTION

A. Collateral Ligament tension:

Femoral component size(s) and position. It is the combination of femoral component a-p dimension and tibial polyethylene thickness that determines joint line.  Two combinations may be possible and the one that establishes the best joint line height will be preferred.

B. Femoral Component Rotation:

Epicondylar axis is the most reliable indicator

C. Joint line height:

Combined femoral component size and tibial polyethylene thickness that leave the inferior pole of the patella above the joint line wil be preferred.

 

The flexion gap is determined by the position of the posterior femoral condyles and the tibial articular surface. The tibial surface has already been established and the posterior articular surface of the femoral condyles will be determined by the size of the femoral component and how it is attached to the femur.

A.  Rotation of the femoral component should be parallel to the epicondylar axis. Palpation of residual posterior condylar bone after the removal of the failed component will help determine if ithad been placed in internal rotation.

B. Choose a femoral component that will be big enough to stabilize the knee in the flexed position, without concern for fixation or bone defects. This is important. If intramedullary rods are used they will determine the anterior-posterior position of the component and consequently the tightness of the flexion gap.

Assess the gaps that remain between the interface side of the posterior femoral component and the remaining bone on the posterior femur. Gaps should be reconstructed with augments. Do not apply the posterior femoral component directly to a deficient surface. This leaves the knee unstable in flexion and displaces the extensor mechanism anteriorly. Failure to support the flange posteriorly has however been implicated in femoral component loosening. (King & Scott)

C.  The combination of femoral component and tibial polyethylene will establish the joint line. Choose the pair that stabilize the knee in flexion and that keep the inferior pole of the patella above the joint line, if possible.

 

Step 3 Balance the Extension Gap

The final major variable to be solved in the reconstruction is the position at which the femoral component will be seated to create an extension gap that equals the flexion gap. A space between the tibia and femur in extension that is larger than the flexion space can be diminished by attaching distal femoral augments to the femoral component. Asymmetric bone defects, or distal femoral cuts that were cut with the wrong angle originally can be corrected with augments on only one side, or larger augments on one side than the other. Correct valgus alignment is ensured with press fit medullary stems if the entry point in the distal femur has been selected correctly and the stems are large enough to make endosteal contact in the femur, yet not so large as to be deflected by asymmetry in the bone.. Within limits, a relatively tight extension gap can be enlarged by resecting additional distal femur. In general terms, there is usually scant bone to spare, and resection of distal femur creates problems with patella baja, extensor lag and ultimately with the attachment of the collateral ligaments.

Conventional ligament releases may be required at this point. If so, it will be necessary to return to Step 2 to confirm that there is not a need for a thicker polyethylene insert as a result of the releases.

 

Critical Decision Points

Two circumstances may arise that force one of two choices upon the surgeon.

1.     Flexion extension instability. In some very difficult reconstructions, the flexion gap sags open and is much more spacious than the extension gap. It will not be possible to find a femoral component large enough in the AP dimension to stabilize the knee in flexion- such a component would overhang tremendously medial to lateral. If an inordinately thick tibial insert is selected for these knees, it will not be possible to extend the knee. This knee will require a constrained condylar prosthesis or a ligament reconstruction, transposing the medial collateral ligament anteriorly on the femur for example to tighten the knee selectively in flexion

 

2.     Valgus instability. When no amount of release of the lateral structures can place the medial structures under tension, then progressively thicker polyethylene inserts will only create a flexion contracture. This calls for a constrained condylar insert or a proximal advancement of the medial collateral ligament on the femur, with or without an augmentation. Ligament reconstructions are required only rarely.

Choices

1. Constraint  in the prosthesis

2. Ligament  reconstruction and or advancement. These are required very rarely, should be regarded as experimental and are never a substitute for conventional ligament releases.

Treatment for Specific Problems

The stiff knee

     Whether lacking flexion, extension or both, the stiff knee may be one of the most dismaying and difficult knees to revise. The problem will be unlikely to respond to revision if the knee had poor motion prior to the original arthroplasty. Arthroscopic release of intra-articular fibrosis, followed by manipulation and physical therapy has been of modest value in some patients with stiff knee arthroplasties.  Manipulation alone, after 6 weeks, once fibrosis has occurred is unlikely to help, and risks disrupting the extensor mechanism or fracturing the femur.

Mechanical impediments- Potential for Revision

 

Poor flexion

1.  Non surgical

*    Inability to comply with physical therapy

*    Aggressive inflammatory collagen disease

*    Contracted extensor mechanism may not respond to further surgery.

2.  Surgical

 

Poor extension

i.   Tight flexion gap

ii.       Tight PCL

*    The PCL may be tight despite good component position. Recession, or partial release of the PCL is recommended if the PCL displaces the tibia anteriorly during flexion.

iii.  Posterior condyle osteophytes

*    Anterior tibial tilt tightens collateral and posterior cruciate ligaments as the knee flexes.

iv. Proximal joint line migration

*    Proximal joint line migration, resulting most commonly from correction of flexion contractures by resection of distal femur, causes stiffness by compromising PCL  and collateral ligament mechanics.

v.  Malrotation of components (especially the femoral)

*    Rotational malposition of the femoral or tibial components, though difficult to diagnose, may be the basis for poor motion. Malrotation jams the components together painfully during flexion, causing stiffness and contributes to patellar maltracking.

Other Considerations

i.   Tight extension gap Resection of additional distal femoral bone may be an important part of revision surgery.

ii.   Tight posterior structures

iii.  Quadriceps weakness and failure to comply with physical therapy

iv. Consider spastic disorders


 

References:

*    General

1.     Vince KG: Revision Knee Arthroplasty in Chapman: Operative Orthopedics 1993

2.     Vince KG and Dorr LD: Revision Total Knee Arthroplasty for Aseptic Failure. Technique. in Orthop.1:83-93, 1987

3.     Vince KG: Revision Knee Arthroplasty. Instructional Course Lectures;1993

4.     Revision Knee Arthroplasty Manual by  Booth R., Insall JN and Vince KG.(published by the Zimmer company)

5.     Vince KG: Limb Length Discrepancy after Revision Total Knee Arthroplasty. Techniques. in Orthop. 3:35-43, 1988

6.     Whiteside LA:  Cementless reconstruction of massive tibial bone loss in revision total knee arthroplasty.  Clin. Orthop. 248:80-6, 1989.

*    Mechanisms of Failure

1.     Alexiades M, Sands A, Craig SM et al:  Management of selected problems in revision knee arthroplasty.  Orthop. Clin. No. Amer. 20(2):211-9, 1989.

2.     Buchholz HW, Heinert K:  Long term results of cemented arthroplasty.  Analysis of complications fifteen years after operation.  Orthop. Clin. No. Am. 19(3):531-40, 1988.

3.     Cameron HU, Hunter GA:  Failure in total knee arthroplasty: mechanisms, revisions, and results.  Clin. Orthop. 170:141-6, 1982.

4.     Insall JN: Total Knee Arthroplasty, in Insall JN, ed. Surgery of the Knee. Churchill Livingston 1984

5.     Kaufer H, Matthews LS:  Revision total knee arthroplasty: indications and contraindications.  Instr. Course Lect. 35:297-304, 1986.

6.     Moreland JR:  Mechanisms of failure in total knee arthroplasty. Clin. Orthop. 226:49-64, 1988.

7.     Schneider R, Abenavoli AM, Soudry M et al:  Failure of total condylar knee replacement.  Correlation of radiographic, clinical, and surgical findings.  Radiology 152(2):309-15, 1984.

8.     Scott WN and Rubinstein M. Failure Rate of Primary Total Knee Replacement. in Total Knee Revision Arthroplasty ed Scott WN, pp1-8, Grune and Stratton, New York 1987.

9.     Vince KG and Dorr LD: Surgical technique of Total Knee Arthroplasty: Principles and Controversy. Techniques in Orthop. 1:69-82, 1987

10.            Windsor RE, Scuderi GR, Moran MC et al:  Mechanisms of failure of the femoral and tibial components in total knee arthroplasty. Clin. Orthop. 248:15-9, 1989; discussion 19-20.

*    Infection

1.     Bindelglass DF, Cohen  JL and Vince KG: The Infected Total Knee Arthroplasty. Am J Knee Surg. 4: 94-105, 1991

2.     Jerry GJ Jr, Rand JA, Ilstrup D:  Old sepsis prior to total knee arthroplasty.  Clin. Orthop. 236:135-40, 1988.

3.     Darouchie RO, Hamill RJ, Musher DM et al:  Periprosthetic candidal infections following arthroplasty.  Rev. Infect. Dis. 11(1):89-96, 1989.

4.     Levine M, Rehm SJ, Wilde AH:  Infection with Candida albicans of a total knee arthroplasty.  A case report and review of the literature.  Clin. Orthop. 226:235-9, 1988.

*    Radiographic and Scintigraphic Evaluation

1.     Duus BR, Boeckstyns M, Kjaer L et al:  Radionuclide scanning after total knee replacement:  correlation with pain and radiolucent lines.  A prospective study.  Invest. Radiol. 22(11):891-4, 1987.

2.     Hendrix RW, Anderson TM:  Arthrographic and radiologic evaluation of prosthetic joints.  Radiol. Clin. No. Am. 19(2):349-64, 1981.

3.     Hunter JC, Hattner RS, Murray WR et al:  Loosening of the total knee arthroplasty: detection by radionuclide bone scanning. Am. J. Roentgenol. 135(1):131-6, 1980.

4.     Rosenthall L, Lepanto L and Raymond F. Radiophosphate Uptake in Asymptomatic Knee Arthroplasty. J Nucl. Med 28: 1546-1549, 1987.

5.     Schneider R, Abenavoli AM, Soudry M et al:  Failure of total condylar knee replacement.  Correlation of radiographic, clinical, and surgical findings.  Radiology 152(2):309-15, 1984.

6.     Hofmann AA, Wyatt RW, Daniels AU et al:  Bone scans after total knee arthroplasty in asymptomatic patients.  Cemented versus cementless.  Clin. Orthop. 251:183-8, 1990

*    Femoral Component Loosening

1.    King TV, Scott RD:  Femoral component loosening in total knee arthroplasty.  Clin. Orthop. 194:285-90, 1985.

---

Previous Lecture  Index  Next Lecture